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In Vermont there is an abundance of runners who tough out the hilly roads and muddy trails year round, but there comes a time when even the some of the toughest runners around transition inside and onto a treadmill. Spending significant time on a treadmill sometimes leads to injury as there is an increased load on the Achilles tendon. 1 Another reason is that runners will tend to ‘mix it up’ more on a treadmill to keep from getting bored. This means more hill work, more speed work, etc. which can place the tendon under a new and different type of strain (eg. not the exact type of strain that it has adapted to absorb). 2
So, if you take to the treadmill a bit during crummy weather be on the look out, and try an ‘ounce of prevention’.
Who Gets it?
Achilles Tendonitis (AT) most commonly effects running athletes in their 30’s and 40’s. Males are affected more often than females accounting for 89% of all reported Achilles tendon injuries.
On average, 50% of runners will experience Achilles tendon pain at some point in their careers. And, for some, it can become a reoccurring nightmare.
What is it?
Achilles Tendonitis describes an irritation of the tendon along the back of the ankle that connects the calf muscles to the heel.
The AT is the thickest, strongest tendon in the body. The reason for this is that virtually all of the force generated when you “toe off” the ground during running is transmitted through the Achilles – and this force can be as much as three times your body weight!
‘Tendinopathy’ describes an accumulation of micro-damage that results when the demands placed on the tendon exceeds its functional capabilities:
LOAD X REPS > RECOVERY
Wear and tear builds up and the tendon may become inflamed (“Achilles Tendinitis”). Often, however, the tendon becomes dysfunctional with minimal inflammation – in these cases we see a ‘failed healing’ response and a gradual degeneration (not inflammation) of the tendon occurs. 3 For this reason, medical professionals will often refer to it as Achilles Tendinosis (remember ‘itis’ mean inflammation).
In short – the bodies’ natural healing mechanisms are unable to keep up with the daily wear – degeneration, micro-tearing and pain results.
The bulk of this blog is focused on chronic, overuse tendinosis; however, an Achilles injury can occur acutely. The severity of acute injuries is graded based on the amount of damage to the tendon:
Grade I: Mild strain, disruption of a few fibers. Mild to moderate pain, tenderness, swelling, stiffness. Expected to heal normally with conservative management.
Grade II: Moderate strain, disruption of several fibers. Moderate pain, swelling, difficulty walking normally. Expected to heal normally with conservative management.
Grade III: Complete rupture, often characterized by a “pop,” immediate pain, inability to bear weight. May require surgery to repair.
Here is an example of an ‘Acute’ injury in a world famous footballer David Beckham. He experiences a complete rupture of the Achilles tendon while playing for AC Milan in 2010. Obviously this ‘single episode’ acute injury more likely represents the culmination of sub-clinical trauma accumulation that weakens the tendon. How else can we explain a seemingly benign movement resulting in a torn tendon in a well-conditioned athlete? This injury happens in the very final minutes of the game – presumably fatigue plays a role as well. Beckham described afterwards that he had been training furiously to try and keep his spot on the English National Team for one last World Cup go. Failure to calculate appropriate recovery into training is a hallmark of this type of injury.
Encouragingly – Beckham was able to return to the pitch (as a substitute) only 6 months after surgery.
* Side note – watch Beckham’s reaction immediately after the injury. He turns and swings an arm behind him to swat the player that just kicked him… only, there’s no one there. Victims of Achilles tendon ruptures almost always report thinking that they were kicked (or shot!).
As wear and tear builds up, and use overpowers recovery, the body’s healing and remodeling mechanisms are unable to maintain a healthy tendon. On a cellular level, what we see is that some of the collagen fibers are ruptured (If a tendon is a rope, think of collagen fibers as being the individual strands that make up the bulk of the rope). The body is able to lay down new fibers to replace the damaged ones, but it does so in a ‘disorganized’ way. These new fibers do not ‘align’ themselves properly and thus have limited ‘tensile’ strength.
A useful metaphor: Healthy Tendons – visualize a box of uncooked spaghetti noodles. All fibers are tightly packed together and lined up to resist forces in a single direction. This image is of a healthy tendon under a powerful microscope:
Unhealthy Tendons more closely resemble a bowl of cooked noodles. In the image below (which is of a damaged Achilles tendon) you can see the fibers are disorganized (not properly aligned in the plane of applied stresses) and not tightly packed together. This tissue is not ready to transfer tensile forces of 3 x your body weight!
What Does it Feel like?
The most common site to feel pain and stiffness is in the mid-portion of the tendon, just above the heel, although it may also present at the insertion onto the heel bone.
Early in the onset, the pain may be most obvious in the morning and at the beginning of activity (before getting properly ‘warmed up’). As the injury progresses, often the pain will be present towards the end of exercise (when fatigued) and/or after activity (when inflammation settles in).
The athlete may also experience increased calf muscle tension or a tendency to cramp. Additionally, there may be obvious swelling in the tendon, or at the insertion of the tendon onto the bone.
*** The ability of the athlete to ‘run through’ the pain initially often leads to a delay in seeking help. However, as we will see, treatment is more effective when caught early, so heed early warning signs. (David Beckham reported having very little awareness of pain/stiffness prior to his rupture… a ‘little’ pain and stiffness can indicate big problems on the horizon! ***
How to Diagnose it?
An accurate diagnosis is often possible in the office, and a proper clinical assessment can effectively rule out a tendon rupture. 4
Imaging techniques are often not needed to diagnose AT.
However, in difficult cases where the presentation is not completely clear, an MRI or Ultrasound may be helpful. 5
A evaluation with a sports physical therapist should include a thorough examination of the foot and ankle, a detailed movement assessment (e.g. your ability to walk/run, squat, stand on one leg, etc.) and an in-depth account of the injury onset and training history.
Your Therapist will need to rule out other potential causes of posterior heel pain such as sural nerve entrapment/irritation, bursitis, and Posterior Ankle Impingement (PAI). PAI is often mis-diagnosed as ‘Insertional’ AT as it mimics many of the same symptoms. In this case, the pain and tissue damage is caused by compression of soft or bony tissue between the shin bone (tibia) and the heel bone (calcaneus). This can lead to inflammation in the tendon or the ‘Bursa’ sacs surrounding the tendon near the heel.
Some people have a small extra bone in the back of their ankle called the Os Trigonum that can lead to impingement. Also, small fragments of bone (i.e. bone chips) can break off the ankle bone and contribute to the development of an impingement syndrome.
PAI may eventually lead to an Achilles Tendonopathy, but in these cases, the underlying cause, pathology and treatment strategy is different.
For more information on posterior ankle impingement click hereor here.
What Causes it?
Achilles tendinopathy may result from a combination of several different variables, including:
Ankle joint stiffness
Calf muscle tightness
Calf muscle weakness
Abnormal foot structure
Asymmetry in lower limb mechanics (when running)
Training errors (*see below for examples)
What to do?
If you are beginning to feel symptoms of AT, be as proactive as possible. Remember, by the time you are feeling these symptoms, a sub-clinical accumulation of micro-trauma has likely been in place for weeks or months. It is not a new injury, it is simply an injury that has just recently reached the ‘tipping point’ and become symptomatic. Thus, it is unlikely to resolve on it’s own. If you rest it a bit, you may notice a resolution of symptoms. But, if you wish to return to prior training intensities, the underlying factors that contributed to the onset must be addressed.
Do not let symptoms linger unaddressed!
The longer the duration of symptoms, the more difficult the recovery.
Some Basic Strategies to Try on Your Own Immediately
1) If you cannot walk quickly for at least 30 minutes (without a limp!) you should absolutely not be running. If your symptoms have reached this level of severity, some pro-active rest is necessary. Consult with a Sports Physical Therapist.
2) If you can walk longer distances, and run short distances without pain or limping, and you plan to continue running – reduce your weekly volume by 30-40%.
3) Warm up well! This may include a dynamic warm up, the use of non-impact machines (if running at the gym), or walking for 1-2 minutes and then transitioning to a running pace gradually. In the winter months, I’ve instructed outside runners to soak their legs in a hot bath / hot tub for 4-5 minutes just prior to running. The use of a compression sleeve to maintain muscle temperature after this type of ‘warm up’ can be helpful.
* See Video for some ‘dynamic warm up’ strategies.
4) Start stretching your calfs – Some light / dynamic stretching pre-run and some longer static stretching post-run.
* See Video
5) Start Foam rolling your calfs – This will decrease the muscle stiffness and allow for more efficient lengthening throughout your push off.
6) Avoid running on a road that is banked so that the center is raised relative to the sides (unless you can run down the center). Avoid training on a track and running in the same direction every session (this is especially important for indoor / short tracks).
7) Immediately stop any hill or speed work.
8) Increase your low-impact training – A spinning bike or Stairmaster are potentially good choices. Pool running is a great choice as well.
9) Consider your shoes – Have you recently made changes in shoe type? Both increasing or decreasing the amount of support (or stability) in the shoe can change the stresses placed on the AT. If you have not changed shoes in 4-6 months, perhaps it’s time. 300-400 miles may be the limit for one pair of shoes depending on body type and running style. (Best to consult with a Sports Medicine Professional familiar with running biomechanics for this one.)
10) Begin a training log. This should include daily information of training volume, intensity, and information on behavior of symptoms. A few weeks worth of this information can be very valuable to a Sports Medicine Professional counseling you on a proper recovery.
Can this be Prevented?
Yes. Like so many repetitive stress injuries, the onset of AT is related to overuse, misuse and/or abuse. See the table below to illustrate some potential contributing factors.
The most effective prevention strategy is to:
1) Have your training program / routine evaluated – For relatively new runners this means insuring that your progression of volume is not greater than 10% every 2 weeks. Additionally, your training days and rest days should be distributed appropriately throughout the week and your training ‘type’ (i.e. volume days vs. speed work or hills) needs to be appropriate for your experience level.
For more experienced or competitive runners this means calculating your acute:chronic training ratio and ensuring proper recovery strategies.
2) Have your gait evaluated – Get a running assessment by a sports Physical Therapist. This running assessment should include a video analysis of your running form (shoe and barefoot), a movement screen, and a detailed strength and mobility screen.
***** Recommendations on ‘Proper’ Running Form should be 100% individualized to the athlete based on body structure, injury history, training history, current gait mechanics and performance goals. Generic advice on ‘proper form’ that is distributed unselectively is not helpful… and may be harmful! *****
* Click herefor more info on our Running Assessment – we offer the most detailed and professional screen in the region!
There are nearly countless potential variables that can increase strain at the Achilles Tendon and contribute to breakdown. Below are only a few examples to highlight the myriad factors that can contribute to the development of AT.
Animal Studies have shown that IASTM results in a quicker recovery from tendon injury, as well as a biomechanically stronger tendon after recovery. 11
Also, IASTM, when added to a comprehensive rehab program for AT, has been shown (in humans!) to provide added benefit – both short term and long term – including greater recovery of function. 12
Joint Mobilization may be indicated if motion restrictions in the foot or ankle are found to increase strain to the Achilles. 13 Mobilization of movement restrictions further up the ‘biomechanical chain’ may also be indicated. 14
A properly tailored exercise program will take into consideration all evaluation findings and will often include:
1) Calf Strengthening. Weakness in the calf has be shown to be risk factor for developing AT. 15
2) Hip Strengthening. Hamstring weakness 16 and gluteal weakness 1718 have been shown to contribute to the development of AT.
3) Core Strengthening. Dysfunctional Activation of the Transverse Abdominus muscle at specific moments during the gait cycle, has been demonstrated in runners with AT. 19
Also, Movement Re-Training Exercises can help to target specific gait dysfunction and improve gait efficiency. Research has demonstrated over and over again that isolated strengthening does not automatically translate into improved motor patterns (or improved running mechanics). A comprehensive rehabilitation strategy must include exercises specifically tailored to ‘re-educate’ proper movement.
Also, athletes with chronic AT have been found to have weaker Hip/Gluteal muscles when compared to their uninjured counterparts. 20
The exercise prescription should also include a graded progression of exercises prescribed to influence cellular level changes in the Achilles tendon. 21
This may include high-rep, low-rep exercises to increase circulation and improve lymph drainage. 22
Alternatively, this may include more aggressive ‘eccentric’ loading exercises for the more chronic/degenerative tendon. The goal of this type of intervention is to strengthen the tendon, reduce inter-tendon fluid retention / tendon thickness, and perhaps mitigate the negative effects of ‘neovasculization’ (the formation of microvascular networks that infiltrate a degenerative tendon and bring along nerve endings that may contribute to pain perception.) 23
* In patients with mid-tendon pain and swelling (the most common type of AT) a progressive eccentric loading program has been shown to be 90% effective *24252627
Not only that, an aggressive eccentric exercise based approach has been shown to be effective even in chronic cases when other, more traditional (but ineffective!) approaches had failed.
A thorough evaluation of foot and ankle mechanics may lead to the recommendation for custom foot orthotics. Corrective orthotics may be indicated if:
1) Poor alignment of the foot, relative to the ankle, is found (a common finding in athletes with AT) 2930
Orthotics are not necessary in most cases, but when needed, can help significantly.
Improving the foot stability through corrective orthotics can be effective at reducing pain 3133 Also, orthotic therapy has been shown to improve the ‘load tolerance’ of Achilles Tendons in runners. 34
Corrective orthotics are a potentially effective adjunct to a comprehensive approach.
*** The Sports Physical Therapists at VASTA can fabricate custom orthotics if indicated.***
Low Level Laser Therapy (LLLT).
LLLT has been shown to have a positive effect on recovery from tendon injury. 35
With respect to AT specifically, some controlled studies have demonstrated reduced inflammation and decreased pain 36 as well as accelerated healing rates. 37 But, others studies have failed to demonstrate effectiveness. 38
There is certainly a theoretical model that supports it’s use as laboratory studies of LLLT has demonstrated a reduction of inflammation and an effectiveness equal to NSAIDS in animal studies. 39
While the research is not overwhelming, negative effects are limited and costs are minimal. LLLT can be used as an adjunct to a comprehensive approach to both acute and chronic AT.
While a heel lift may be useful in cases of acute AT, where a proper gait pattern is difficult due to pain, the usefulness in Chronic cases appears to be minimal. 4041
Research into the effectiveness of night splints for AT is mixed. While some studies have shown a modest positive effect at reducing pain 42 others have demonstrated minimal effect.
For a patient who is compliant with a proper exercise based rehabilitation program, there appears to be no additional benefit to using a night splint. 4344 Given that night splints are uncomfortable and often interrupt sleep (when the body is in ‘repair mode’), we typically do not recommend the use of night splints.
Iontophoresis is the use of a medicated pad connected to an electric current to ‘drive’ the ionized medication into the tissue. Iontophoresis can be used to delivery a corticosteroid without injection. Compared to injection, it is safe, and without the risk of tendon rupture 45
Some studies have indicated a potential positive effect with Iontophoresis in patients with AT 46 but it appears to not be as effective in chronic cases.
Corrective Exercise, Manual Therapy and Training Modifications for Load Management are all part of a comprehensive rehabilitation plan.
It is important to give this time before considering other options – say, four to six months. This is due to 2 main considerations: 1) Tissue Response Time. Making training modifications and beginning interventions (like say, eccentric loading) in an attempt to positively effect tissues metabolism and micro-structure, is time dependent. It will take time for the cumulative effect of these changes to result in a healthier tendon and a meaningful reduction of pain; and 2) There isn’t convincing evidence that other options are any better. There are many more aggressive surgical and ‘alternative’ options out there. However, these are often expensive, sometimes very painful, may carry adverse side-effects and are not proven to be any more effective (and in many cases have been proven to be less effective) than the Physical Therapy approach described above.
For informational and decision making purposes, additional information is provided below regarding more invasive and ‘alternative’ options.
For patients looking for more information about minimally invasive and surgical interventions VASTA works in close communication with area Physician Specialists.
*** Talk to your Physical Therapist about setting up a referral ***
The effectiveness of acupuncture in the treatment of AT is questionable as studies provide mixed results. Additionally, most studies that report some positive effect are, for the most part, of poor design and of very ‘low power’ (eg. single ‘case studies’ or non-controlled designs of small sample sizes), making it difficult to make meaningful recommendations.
Some recent research on Electro-Acupuncture has been interesting, 47 but certainly more research is needed as the ‘jump’ from animal studies to proving real significant improvement in humans beings is a big one.
* Recommendation – Not routinely recommended, but not necessarily discouraged (as an adjunct to rehabilitation) for patients who have had a positive response to acupuncture in the past.
Extracorporeal shock wave therapy (ESWT)
ESWT is the therapeutic use of abrupt, high amplitude pulses of mechanical energy, similar to soundwaves, generated by an electromagnetic coil. The research looking into the effectiveness of ESWT for AT has produced mixed results. Much of the research in support is not well controlled and overall the research supporting it’s use is unconvincing. 48
While some studies have shown a potential therapeutic effect, this is inconsistent. In addition, in studies that have demonstrated effectiveness, the therapeutic effect is no better than exercise therapy, 49 maybe less so. 50
* Recommendation – Not Recommended. It is expensive, and shown to be of no added benefit when compared to a proper rehabilitation approach.
Nonsteroidal Anti-Inflammatory Drugs (NSAIDS)
NSAIDs, in addition to having multiple deleterious ‘side’ effects, have been shown to have minimal>no positive effect on recovery for patients with AT. 5152
* Recommendation – Not Recommended.
The effectiveness of Corticosteroid Injections is questionable. Some studies have shown 53 short term pain relief, but likely at a cost of negative long-term effects including increased likelihood of recidivism, 54 and the proven adverse effect of delayed healing. 55
‘Peritendonous’ injections have been recently advocated as an adjunct to a proper, comprehensive conservative rehabilitation program due to less deleterious tendon weakening effects, 56 however, the positive effects remains somewhat equivocal. 57
* Recommendation – Direct Tendon Injections are not recommended. Peritendon Injections can be considered if the patient has failed to respond to an aggressive and comprehensive rehabilitation approach (4-6 months), and is determined to be a good candidate for this procedure.
Glyceryl Trinitrate (GTN) Patches
Also known as Nitroglycerine Patches, GTN is more commonly used to treat chest pain associated with heart disease. When Nitroglyercine is metabolized by your body it releases nitric oxide (NO) which, among other things, is a vasodilator (relaxing blood vessels and improving local blood flow).
NO has been shown in Laboratory studies to aid in Tendon healing (when your tendon is healthy, native NO levels are quite low, but when injured, its production is ramped up).
In theory GTN patches work by stimulating collagen synthesis (the type of fibers that make up a bulk of the AT). The research on GTN patches is somewhat mixed. Studies have determined a positive potential effect for tendonitis (in general), 58 and in patients with AT in particular 59 however, other studies looking specifically at AT have provided conflicting results 60 questioning it’s usefulness.
Some researchers also question the mechanism of effect in cases where a benefit is demonstrated. There is uncertainty whether the tendon healing effect of NO is real, or if the patch may simply act to temporarily decrease pain by increasing blood flow, but not provide any meaningful benefit to aid true structural healing and recovery. This is not a universally shared opinion. 61
Also, GTN patches also carry some concerns for adverse side effects, including severe headaches 62, and should be used with caution and always under the supervision of a medical provider.
* Reccommendation – Not recommended for widespread use. May be considered, under medical supervision, and as an adjunct to rehabilitation for patients who have failed to respond to an aggressive and comprehensive rehabilitation approach (4-6 months).
Sclerotherapy involves injecting a chemical (typically polidocanol) into a blood vessel, which results in the sclerosis of that vessel. The reasoning behind using sclerotherapy for AT is based on the finding that there is a proliferation of small blood vessels in areas of chronic tendinopathy (see discussion of ‘neovascularization’ above). Importantly, small nerve endings appear to travel along with these new blood vessel formations. One theory is that these nerve fibers are a major pain generator in patients with AT. It is thought that the sclerosing therapy could not only destroy the vessels, but may also eradicate the pain-generating nerve fibers. A musculoskeletal specialist physician performs these injections under ultrasound guidance.
The research investigating sclerosing injections have shown some promise to reduce pain in more chronic cases that are unresponsive to conservative measures. 636465
Although some studies suggest that polidocanol injections appear to provide pain relief, it is unclear what role they may play in promoting tendon healing. Other studies have failed to demonstrate improvement and specifically called into question prior studies claiming positive effects. 66
Last, and perhaps most importantly, long term studies must be done to demonstrate that the procedure is not ultimately detrimental to true healing. 67
* Recommendation – Not recommended for widespread use. The theory is intriguing, but more research must be done to demonstrate that this invasive technique provides added benefit over conservative approaches and does not weaken the tendon and expose it to future injury.
Prolotherapy is generically defined as an injection of an irritant solution (e.g. dextrose) into ligaments or tendon insertion in an effort to relieve pain and promote a local healing response. Overall Prolotherapy is a generic term with a wide range of meanings as the substance injected and the mode of injection can vary significantly.
For that reason, it is difficult to generalize about the data supporting or refuting it’s usefulness. With consideration of AT in particular, the research is mixed. Some studies have shown a positive effect (decreased pain levels and improved tendon ‘structure’) for patients with chronic AT. 68
Other studies, however, have shown only modest short-term improvements when compared to even a limited exercise-based rehabilitation program, with no added benefit seen in the long term.69
Also there is some potentially valid anecdotal concern that “prolotherapy injections may indeed help with protein synthesis and collagen formation… but (it may be) of (the) softer less dense Type III collagen which is formed rapidly and quickly after the injections, which for a stabilising ligament isn’t the best type… this excessive Type III collagen produced by prolotherapy injections may well actually prevent the formation of normal Type I collagen from being laid down with normal natural healing mechanisms, and so could cause potential long term deficits.” 70
* Recommendation – Not recommended for widespread use. Given the significant cost of the procedure (it is not covered by insurance), the painful nature of the procedure, and the questionable added benefit, it’s indication is uncertain. However, serious adverse side effects are very limited 71 with infection being the top concern. Thus, in some limited cases, where speed of recovery from a new injury is the top priority (eg. professional sports), prolothery could be considered, but the added benefit to an aggressive and comprehensive rehabilitation approach is questionable.
Platlet-rich Plasma (PRP) Injections
The material utilized in PRP injections is typically blood plasma with a high concentration of autologous (harvested from you) platelets. This PRP solution contains several different ‘growth factors’ and other ‘cytokines’ that are known to stimulate healing in soft tissues.
PRP injections are thus advocated as a ‘pro-regeneration’ intervention for damaged and degenerated tendons that have demonstrated a ‘failed healing’ response.
One issue with this theory, however, is that growth factors are already found in elevated concentrations in damaged tendons (even chronic injuries) as this appears to be one of your bodies natural responses to injury. Simply increasing these concentrations more is not a guarantee that the underlying imbalance of degeneration vs. regeneration will be corrected.
However, on a theoretical level, the potential of PRP and ‘regenerative medicine’ in general, is hugely attractive.
Researchers have attempted to investigate the potential effectiveness of PRP with controlled studies. As is almost always the case with a relatively new treatment approach, the initial results are mixed. 72
Despite a strong theoretical model of therapeutic potential, studies have generally failed to demonstrate that PRP injections are more beneficial than saline (placebo injection) in other types of Tendonopathy. 73 No strong evidence exists to suggest it’s usefulness in AT specifically. A recent review of research (a meta-analysis) of PRP use stated that current evidence suggests it “did not provide significantly greater clinical benefit versus placebo or dry needling for the treatment of tendinopathy.” 74
In spite of the limitations listed above, research into regenerative medicine is largely considered to be in it’s early stages. Additional studies are needed to determine the exact usefulness, optimal chemical compounding, optimal dosage, and optimal delivery. However, the potential for ‘regenerative medicine’ is largely considered to be substantial.
* Recommendation – Can be considered for patients who have been unresponsive (4-6 months) to an aggressive and comprehensive rehabilitation approach.
Stem Cell Therapy (SCT)
SCT largely fits under the umbrella of ‘regenerative medicine’ and is still fairly experimental. The basic idea behind this type of therapy is that stem cells can be delivered into an injury tissue (such as a degenerated tendon) and then triggered to differentiate into (turn into) into whatever cell type is needed to enhance the repair process.
While exciting potential exists for SCT, it shares many of the same challenges and questions as PRP treatments. Optimal Stem Cell type, concentration, and mode of delivery have not been well established; and the science demonstrating long term effectiveness and safety is limited. 75
* Recommendation – Not recommended for widespread use.
Surgery is often considered an option of last resort in the treatment of AT. Surgical consideration should be saved for patients where pain persists after exhausting all conservative options.
Surgical Interventions for AT include “minimally invasive” procedures such as Percutaneous Needle Tenotomy (PNT). PNT is performed under ultrasound guidance (to identify the region of degeneration). A small needle is introduced into the damaged tendon region(s) and multiple ‘needlings’ are performed to cause micro-damage in an attempt stimulate a refreshed healing response.
PNT demonstrates reduces complications when compared to open surgical techniques, and it’s use does not preclude or otherwise hinder future surgical intervention if needed. 76
Overall, research demonstrating long-term effectiveness is still scarce, as this procedure is relatively new. One study of PNT for AT patients reported a ~ 75% success rate 77 (but keep in mind this is the ‘fairly good’ recovery rate after 18 months!).
An open surgical debridement (“cleaning up”) is another more invasive (compared to PNT) option. The effectiveness of a surgical debridement for AT appears to be dependent on the extent of damage present. 78
One study of patients with chronic paratendonitis who underwent surgical debridement reported a ‘good’ (or better) success rate ~ 85% of the time. 79
In this same study, patients with true tendon degeneration experienced less encouraging results (‘satisfactory’ results or better ~ 65% of the time).
* Keep in mind! The research definition of ‘good’ or ‘satisfactory’ is rarely what I hear my patients saying they are looking for as an outcome! (eg. it ain’t running pain-free).
Open surgical ‘removal of pathology’ is typically a last resort. Published “success” rates for pain reduction are ~ 80%. The ability of open surgery to improve function again depends on the extent of tissue impairment. With success rates for patients with marked “intratendinous lesion(s)” reported as 54%. 80
This image shows an open ‘repair’ of an achilles tendon. Note the central degenerated section within the tendon.
* Keep in mind! ‘Success’ was defined as a recovery of function. This improved function is typically tested with somewhat easy tasks such as stair climbing and heel raises – not sprinting, jumping, etc.
Also, of interest, a recent review of research has demonstrated that ‘success rates’ published have been worsening over that past few decades (despite advances in surgical techniques). This is believed to be a result of improved research methods demonstrating more realistic reporting. 8182
* Recommendation – All surgical approaches are to be avoided unless absolutely necessary! We are lucky to live in an area with highly skilled surgeons. Should we need their services, we are grateful for their help… but, we should be working hard to NOT seek their help (and they would encourage that!)
For patients looking for more information about minimally invasive and surgical interventions VASTA works in close communication with area Physician Specialists.
*** Talk to your Physical Therapist about setting up a consultation ***